These are best treated as auxiliary hypotheses rather than primary mechanism, as the experimental support is narrower than for the VEGFR2-eNOS axis
Four large trials that studied the effect of intensive glucose lowering on CV risk enrolled few patients with HF and HF hospitalization was not a primary end point [28-31]
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Noteworthy, synergistic effects, while biologically plausible and supported in some settings, may not manifest uniformly in every population or under every protocol
